Part:BBa_K5005005:Design
Primordial plasmid before point mutation.
- 10INCOMPATIBLE WITH RFC[10]Illegal XbaI site found at 5150
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Illegal SpeI site found at 4780 - 12INCOMPATIBLE WITH RFC[12]Illegal NheI site found at 5156
Illegal SpeI site found at 4780
Illegal NotI site found at 6646 - 21INCOMPATIBLE WITH RFC[21]Illegal BglII site found at 3111
Illegal BglII site found at 6045
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Illegal BglII site found at 8312
Illegal BglII site found at 8378
Illegal BglII site found at 8419 - 23INCOMPATIBLE WITH RFC[23]Illegal XbaI site found at 5150
Illegal XbaI site found at 5659
Illegal SpeI site found at 4780 - 25INCOMPATIBLE WITH RFC[25]Illegal XbaI site found at 5150
Illegal XbaI site found at 5659
Illegal SpeI site found at 4780
Illegal NgoMIV site found at 7164
Illegal NgoMIV site found at 8197 - 1000INCOMPATIBLE WITH RFC[1000]Illegal BsaI site found at 3093
Illegal BsaI site found at 8401
Illegal BsaI.rc site found at 1534
Illegal BsaI.rc site found at 6509
Illegal BsaI.rc site found at 7117
Illegal SapI site found at 451
Illegal SapI.rc site found at 3742
Design Notes
NAMPT was cloned using PCR. Homologous arms play a crucial role in the Gibson assembly for connecting NAMPT with the vector. After obtaining the original pTarget-NAMPT plasmid, NAMPT will undergo site-directed mutagenesis at specific points to validate the impact of different mutations on NAMPT enzyme activity. Additionally, in the pTarget-NAMPT plasmid, NAMPT will be positioned downstream of the T7 promoter for directed evolution experiments on NAMPT. Here, you can clearly know that NAMPT is located downstream of the T7 promoter and can be edited by the editor to introduce mutations, resulting in NAMPT variants.
Source
Constructed in this work.
References
Zhu, X., Liu, H., Chen, L., Wu, C., Liu, X., Cang, Y., Jiang, B., Yang, X., & Fan, G. (2022). Addressing the Enzyme-independent tumor-promoting function of NAMPT via PROTAC-mediated degradation. Cell chemical biology, 29(11), 1616–1629.e12. https://doi.org/10.1016/j.chembiol.2022.10.007